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Cell2004mechanismnr

Discoveries of nicotinamide riboside as a nutrient and conserved NRK genes establish a Preiss-Handler independent route to NAD+ in fungi and humans

Bieganowski P, Brenner C

Key finding

NR is a distinct NAD+ precursor in humans, converted via the conserved NRK1/NRK2 kinases — a route independent of the Preiss-Handler pathway.

Summary

Seminal paper identifying nicotinamide riboside (NR) as a bona fide NAD+ precursor vitamin and defining the nicotinamide riboside kinase (NRK1/NRK2) pathway that converts it to NMN and then NAD+ independently of the classical Preiss-Handler route. Using yeast genetics, Bieganowski and Brenner screened for suppressors of a qns1 NAD+ auxotroph and recovered NR as a salvageable precursor. They cloned the NRK1 gene, then identified the human orthologs NRK1 (NMRK1) and NRK2 (NMRK2), showing both possessed kinase activity on NR. This established a third route to NAD+ in mammals, alongside the de novo tryptophan pathway and the Preiss-Handler route from nicotinic acid. The discovery directly enabled the development of NR as an oral supplement — because the NRK pathway is present in humans, dietary NR could be converted to NAD+ in tissues without requiring deamidation. Every subsequent NR clinical trial traces to this mechanistic foundation.

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